Which of the following may cause hyponatremia in enterally fed patients?

Hyponatremia, a condition characterized by low sodium levels in the blood, can indeed be influenced by various medications and treatments, particularly in patients receiving enteral nutrition. Enterally fed patients, who receive nutrition via a tube bypassing normal ingestion, may have specific risks associated with electrolyte imbalances.

Amphotericin is known to potentially cause renal damage, leading to a condition in which the kidneys are less capable of properly handling electrolytes, including sodium. This disruption can contribute to the development of hyponatremia.

Diuretics and laxatives are commonly used to manage fluid balance and gastrointestinal function, but they can also promote the excretion of sodium from the body. In patients who are already at risk (like those on enteral feeding, who may not be getting adequate sodium in their diet), this increased excretion can result in hyponatremia.

Probenecid, often used in the treatment of gout, can affect kidney function as well, influencing the renal handling of various electrolytes, including sodium. Similarly, thiazide diuretics are a well-known class that can lead to sodium loss through increased urine production.

Considering the roles played by all these medications in affecting sodium levels and kidney function, it's